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  2. During an elimination diet, you generally remove foods known to be common allergens or intolerances. These can encompass gluten-containing grains such as wheat, barley, rye, and sometimes oats ...

  3. List of allergens - Wikipedia

    en.wikipedia.org/wiki/List_of_allergens

    A synthetic food dye used in processed foods like confections, soft drinks, flavoring syrups, condiments and convenience foods in order to create a potent yellow or bright green coloring. Prevalence of allergenicity is unclear but it is the most likely azo dye to cause hypersensitivity and reactions may occur from ingestion or skin contact.

  4. Elimination diet - Wikipedia

    en.wikipedia.org/wiki/Elimination_diet

    There is currently no skin or blood test available to identify the offending chemical(s), and consequently, elimination diets aimed at identifying food intolerances need to be carefully designed. All patients with suspected food intolerance should consult a physician first to eliminate other possible causes.

  5. Non-celiac gluten sensitivity - Wikipedia

    en.wikipedia.org/wiki/Non-celiac_gluten_sensitivity

    Frequency. 0.5–13% [13] Non-celiac gluten sensitivity ( NCGS) or gluten sensitivity [14] is a controversial disorder which can cause both gastrointestinal and other problems. NCGS is included in the spectrum of gluten-related disorders. [3] [4] The definition and diagnostic criteria of non-celiac gluten sensitivity were debated and ...

  6. Food intolerance - Wikipedia

    en.wikipedia.org/wiki/Food_intolerance

    Diagnosis of food intolerance can include hydrogen breath testing for lactose intolerance and fructose malabsorption, professionally supervised elimination diets, and ELISA testing for IgG-mediated immune responses to specific foods.

  7. Fructose malabsorption - Wikipedia

    en.wikipedia.org/wiki/Fructose_malabsorption

    Fructose malabsorption, formerly named dietary fructose intolerance ( DFI ), is a digestive disorder [1] in which absorption of fructose is impaired by deficient fructose carriers in the small intestine's enterocytes. This results in an increased concentration of fructose. Intolerance to fructose was first identified and reported in 1956.

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